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10.1084/jem.20210313 Summary Keywords redox signaling, redox metabolism, Alzheimer's disease, neurodegeneration, Tau, APP Citation Holubiec MI, Gellert M and Hanschmann EM (2022) Redox signaling and metabolism in Alzheimer's disease

Interest in mtCYP2E1 has been increasing steming from its role in disease pathogenesis, linked to its activity and localization [5, 14, 16, 18, 20,21,22]

as an inflammatory mediator in cardiovascular diseases

At the same concentration of Glu-HXM, the sam2/sam2 mutant cells started to grow before the 24-hr timepoint, grew fastest between 24 and 48 hours and ended with a slightly higher efficiency to wildtype (Fig 10A)
