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The study was approved by the ethical committee of Karolinska Institutet (616/03) and informed consent was obtained from the patients(or their parents)

Such behavioral deficits in A53T mice were accompanied by the presence of elevated -synuclein monomers and aggregates (Supplemental Figure 1J) and a remarkable loss of dopaminergic neurons in the substantia nigra, as indicated by tyrosine hydroxylase (TH) staining (Figure 1C)

doi: 10.1186/s12870-020-02445-w 32 GulN.HaqZ

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