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The "Thiol-Lock" Protection: The primary antioxidant mechanism of GSH relies on its reduced state

Having more NAD+ available helps activate sirtuins and other biological systems linked to longevity, supporting DNA repair, cell upkeep, and resistance to stress

This is the mechanism that makes TB-500 relevant to injuries that involve slow or stalled healing: when the local repair response has exhausted its resources, TB-500 recruits fresh cells from the surrounding tissue and circulatory system into the damage zone

Concentrations above 2 percent are rarely necessary and may increase irritation risk without proportional benefit