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Description
In AD pathology, abnormally modified Tau proteins regulate microglial activation: Hyperphosphorylated Tau enters microglia via CX3CR1 receptor-mediated endocytosis, activating Syk tyrosine kinase signaling to induce phagolysosomal dysfunction and pro-inflammatory mediator release (36,37)
J Biol Chem 236: 20852088
The peptide reaches the tissues that need it most without requiring systemic distribution
To reach these drug targets, they must traverse biological environments
