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Cells 39, 6064
Evidence supporting oxidative stress in a moderately affected area of the brain in Alzheimers disease
KLF9 may regulate IR via KLF9/PGC1/TRB-3 signaling pathway and promote hepatic gluconeogenesis and hyperglycemia
They exhibit higher potency and specificity, although their short half-life in the body, cost, and instability are notable drawbacks
