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Conditional deletion of Tim-3 in dendritic cells led to increased accumulation of ROS resulting in Nod-like receptor (NLR) family pyrin domain containing 3 (NLRP3) inflammasome activation

The wounds treated by SeC@PA MN(+) exhibited decreased M1-phenotype macrophage (F4/80 + and CD86 + ) infiltration and increased M2-phenotype macrophage (F4/80 + and CD206 + ) infiltration as compared with the control group and C@PA MN(+) group (Fig

If, indeed, oxidative stress contributes to sleep homeostasis, this manipulation would be expected to manifest as an alteration in the homeostatic regulation of sleep

AMPKAMP-activated protein kinase