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Destabilization of the metal binding site may reduce the metal affinity or binding ability of G93A SOD1, resulting in the pathological aggregation observed in mouse models and humans with this mutation

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Abstract The evidence to date, coupled with advances in immunology and genetics has afforded the field an unparalleled opportunity to investigate the hypothesis that a subset of patients with schizophrenia may manifest an immunophenotype, toward new potential diagnostics and therapeutics to reduce risk, alleviate symptoms, and improve quality of life in both at-risk populations and patients with established schizophrenia