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Reported experiments in dermal fibroblast cultures and rodent wound models have examined its association with collagen synthesis (types I and III), glycosaminoglycan and proteoglycan expression, fibroblast activity, and angiogenesis-related pathways such as TGF-beta signaling

This suggests that GSH deficiency caused an increase in TNF- and decrease in PGC-1 , as well as a decrease in the VDR/GLUT-4 needed for the action of 1,25(OH) 2 VD in muscle

(One important accuracy note: NAD+ is a coenzyme rather than a peptide, but it's central enough to longevity research to belong in any serious overview of the category.) The Four Pathways of Longevity Research The compounds below map to four distinct aging-research pathways: NAD+ metabolism NAD+ Telomere and pineal-gland biology Epithalon Mitochondrial-derived signaling MOTS-c Gene expression and collagen / skin aging GHK-Cu 1