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research established the IGF-1/Akt/mTOR pathway as central to skeletal muscle hypertrophy work done largely with LR3 because native IGF-1 couldn't sustain the receptor activation needed to study chronic effects

Theoretically, availability of GSH as an intact molecule declines when consumed orally, which could explain why oral glutathione was less effective

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Statistically significant association was detected between short IGF1 CA repeats and increased risk for colorectal cancer in hereditary non-polyposis colorectal cancer (Zecevic et al., 2006)