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The eyes after infecting toxoplasma gondii showed accumulation of iron in the neurosensory retina, and increased lipid peroxidation, reduction of GPX4 expression and mitochondrial deformity in the photoreceptor, suggesting that ferroptosis plays a crucial role in the induction of retinochoroiditis during ocular toxoplasmosis (Yamada et al., 2023)

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Similarly, oxidative stress states such as sepsis and critical illness can predispose to pyroglutamate accumulation due to increased GSH demand driven by elevated reactive oxygen species production [65]