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In this section, we focus our attention on the latest investigations describing the role of endothelial NMDARs in the fine regulation of BBB permeability and NVC, i.e., the mechanism whereby an increase in neuronal activity is translated into an increase in cerebral blood flow (CBF) [112,122], and their implication in neurological disorders
To avoid undesired protein aggregation, refolding is usually carried out at low protein concentrations [11]
It will be interesting to decipher whether such mechanisms may be involved in neurocognitive diseases in chronic HIV infection
