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Acknowledgements This work was supported by grants SAF2015-71304-REDT and SAF2016-76520-R from the Spanish Ministry of Economy and Competitiveness
In the context of hepatic IR associated with non-alcoholic fatty liver disease (NAFLD), this molecular pathway becomes dysregulated, leading to impaired suppression of hepatic gluconeogenesis
The mechanism seems to be mediated to gp41 fusion peptide, it has antiviral therapeutic potential (119)
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