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Most studies report an increased sensitivity to experimental pain and, therefore, a decreased pain threshold in depressed compared to non-depressed subjects especially when emotional aspects of experimental pain are taken into account (Ushinsky et al., 2013)

Following 7 weeks of targeted oral L-carnitine supplementation, the trial patients demonstrated a robust, statistically significant increase in peripheral serotonin alongside a marked reduction in standardized physical fatigue scores

the reversible properties of epigenetic modifications posit them as emerging potential targets for next-generation therapeutic interventions [1,2,3,4,5]

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