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GSH is the major cytosolic water-soluble chemical antioxidant and clinical evidence suggests that patients with T2D have a GSH deficiency [8]

33 Understanding the etiological contributors to such donordependent variance might shed light not just on RBC storage biology, but also contribute significantly to our understanding of those pathologies associated with RBC hemolysis due to exacerbated oxidant stress (e.g., sickle cell disease, b-thalassemia, pulmonary hypertension, Down syndrome, inflammaging 39 )

S., Whiteman, M., Yang, H., Jones, D