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It is known in the medical industry as the bodys "master antioxidant" responsible for the detoxification of every cell in the human body

BRL cells were cultured for 24 h in 12-well plates, and treated with 03.5 EU/ml LPS (dissolved on DMEM with 1% FBS) for 24 h

Not at the same dose bioavailability differences make direct substitution pharmacokinetically unsound

FOXO4-DRI acts as a competitive inhibitor of this interaction binding directly to the transactivation domain 2 (TAD2) of p53, displacing FOXO4, and allowing phosphorylated p53 to be exported from the nucleus and directed to mitochondria, where it activates BAX, triggers caspase-3 cleavage (via the p53/BCL-2/Caspase-3 signalling pathway), and drives cell-intrinsic apoptosis