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In connection with the authors current research, we provide a review on the biological function of GST and current developments in GST-targeting drugs, as well as the prospects of future strategies

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Results We show that our model accurately reproduces published clinical and experimental data on the dose-dependent time course of acetaminophen in the plasma, the accumulation of acetaminophen and its metabolites in the urine, and the depletion of glutathione caused by conjugation with the toxic product