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Description
Notably, under BSO treatment, these molecules may increase cysteine availability but should not restore GSH levels, as BSO irreversibly inhibits -glutamylcysteine synthetase (-GCS) the enzyme responsible for GSH synthesis downstream of cysteine 48

Keap1 does not control Nrf1s activity

Optional addition of complementary therapies for enhanced results

This drug reinstated the normal daily dynamics of neurotransmitters in hypothalamic regions important for the production and release of gonadotropin-releasing hormone
