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Lin H-Y, Kuo Y-C, Weng Y-I, Lai I-L, Huang TH-M, Lin S-P, et al
Here, we present experiments addressing the hypothesis that chronic social defeat stress produces changes that allow for increased permeability to ghrelin into regions beyond the ARC and median eminence as a potential mechanism that protects against stress-induced pathology
Such tumors may be compromised with respect to some aspects of mitochondrial biosynthesis and dependent on alternate pathways for growth and/or survival such that synthetically lethal targets emerge
