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Brain response in asthma: the role of lung-brain axis mediated by neuroimmune crosstalk

Nishimura and colleagues showed that loss of ATM in melanoblast stem cells within the hair follicle bulge region in mice could sensitize the stem cells to genotoxic stress, leading to early differentiation, depletion from the niche and progressive hair greying over subsequent cell cycles due to eventual depletion of mature melanocytes from the hair bulb 3,10

Some models of BER show poly(ADP)-ribose polymerase (PARP1) activation during this transient nick and gap phase, which, through the production of poly(ADP)ribose, helps recruit the remaining DNA repair factors, XRCC1, a scaffold protein, DNA polymerase beta and DNA ligase I