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(2015) demonstrated that patients with large-artery atherosclerotic ischemic stroke and transient ischemic attack (TIA) exhibited significant gut dysbiosis, characterized by increased opportunistic pathogens (e.g., Enterobacter, Megasphaera, Desulfovibrio ) and reduced beneficial genera like Bacteroides and Faecalibacterium

Ongoing clinical trials are evaluating other ASO candidates targeting genes such as C9orf72 (e.g., WVE-004, NCT04931862) and FUS (e.g., ION363, NCT04768972), as well as ASOs for other targets like ATXN2 (BIIB105, NCT04494256) and SOD1 (e.g., ISIS SOD1Rx, NCT01041222) (8, 15, 16)

Activation of T cell receptor (TCR) signaling can induce PLCG1 recruitment and activation, followed by elevated intracellular IP3 and DAG levels [55, 56]

We emphasize that reported side effects are predominantly linked to unregulated clinics in certain countries