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& Blaxter, M

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Knocking down ech-6 resulted in amino acid accumulation and inhibited metabolic activity, thereby causing energy crisis and suppressing physiological activities that are dependent on energy homeostasis

Our genetic and pharmacological inhibition of LIPE strongly suggests that lowering unsaturated FA levels by slowing the lipase-mediated lipid degradation process represents a novel therapeutic strategy, a finding that stands in mechanistic agreement with our earlier work on SCD inhibition and augments the relevance of targeting FA metabolism to achieve S homeostasis