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One of the key mechanisms underlying the dark and shrunken morphology of granule cells in the DG during epileptic episodes is excitotoxicity, primarily mediated by excessive glutamate release (Wang and Qin, 2010)

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Observed mechanisms include direct neuronal damage, disruption of key neurotransmitter pathways such as AChE activity, induction of cellular stress responses, and alterations in both locomotor and spontaneous activity patterns, particularly in Drosophila