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Its pro-tumorigenic function is likely mediated through increased OCR and ATP production, which supports cell growth and proliferation [96]

For example, in the study of traumatic brain injury in mice, the lack of GPX1 increased the attack of ROS on hippocampal neurons, causing damage to post-traumatic memory function ( Interestingly, we found in our study that GPX1 is not capable of oxidative stress in all cases

Glutathione supports the livers detoxification pathways directly

Increase in cellular TRX levels may restore the receptor activity and permit the GR to efficiently communicate with target genes
