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Description
Notably, H3K18la has been implicated in multiple immuneregulatory pathways: it directly upregulates PDL1 expression via the POM121/MYC axis on tumor cells, and independently activates transcription of the chemokines CXCL1 and CXCL5, thereby promoting the recruitment of myeloidderived suppressor cells (MDSCs) into the tumor microenvironment, MDSC accumulation, in turn, suppresses CD8 + T cell cytotoxicity and contributes to resistance against ICB (76, 77)
The function of GSH as a reducing agent was dis-cussed in a previous section
Iridoids modulate inflammation in diabetic kidney disease: a review
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