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In the case of Apert syndrome, for example, there is good reason to believe that the causative mutation (in this case, predominantly a 755C-G transversion in the FGFR-2 gene) arises in spermatogonia and is selectively retained in the germinal epithelium because the mutant germ cells enjoy an unspecified selective advantage [4]
Sakurai A, Nishimoto M, Himeno S, Imura N, Tsujimoto M, Kunimoto M, et al
Through a randomized, double-blind, placebo-controlled trial involving 20 pediatric T1D patients, Reddi et al
Our experience shows that this is the single greatest risk to both scientific integrity and safety
