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however, it does not always cause myeloproliferation and can be benign, depending on the specific genetic mutation

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However, despite this potential, translation of ferroptosis to human atherosclerosis has been limited due to inconsistent definition of functional ferroptosis and widespread use of nonspecific oxidative stress markers (that do not confirm iron dependency, selective rescue and membrane phospholipid hydroperoxide executors) to distinguish ferroptosis from other forms of oxidative damage