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Inflammation in adipose tissue is significantly associated with the histological severity of MASLD, with inflammatory processes in visceral adipose tissue potentially preceding and contributing to the progression of the disease to MASH and liver fibrosis [44]

Human enteroids derived from intestinal crypts with history of NEC versus non-NEC were cultured and then subjected to 12 hours of LPS and hypoxia to induce NEC in a laboratory setting

Quantitative determination of uric acid using CdTe nanoparticles as fluorescence probes

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