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Paradoxically, in another study, CD163 was significantly upregulated in symptomatic carotid atherosclerotic plaques and was associated with intraplaque hemorrhage, plaque ulceration, and markers of apoptosis and proliferation, such as activated caspase 3, TUNEL, and Ki67 [117]

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In fact, much of the aging process is a result of the breakdown of tissue repair processes and the subsequent increase in inflammation that occurs when these processes become dysregulated