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Belle A, Laurent V, Pouillon L et al

Meanwhile, ATP signaling via the same receptor enhanced the release of profibrotic tissue-metalloprotease inhibitor 1 (TIMP1), which attenuates the function of ECM-degrading enzymes, resulting in increased interstitial amounts of ECM, a hallmark of lung fibrosis ( 2+ and ATP are well-known stimulating agents of surfactant secretion (Voyno-Yasenetskaya et al., 1991
HCV infection elicits immune responses from the host, including initial innate responses and subsequent adaptive immunity

Proc Natl Acad Sci U S A 120(18):e2221888120
