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In AD transgenic mice, hippocampal mitochondria show reduced Complex I activity, elevated reactive oxygen species, and fragmented morphology even before plaque formation
Comparative analyses further highlight these source-dependent disparities, revealing that the chondrogenic potential of both BMSCs and AD-MSCs in the presence of TGF-3 exceeds that of annulus fibrosus-derived MSCs (AF-MSCs) (Park et al., 2011b)
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