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Several theories have been proposed to explain the mechanism underlying ovarian aging, including free radical theory, apoptosis, telomere shortening, mitochondrial dysfunction, and inflammation theory (Shi et al., 2016
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When membranes function properly, they can efficiently eliminate toxins while protecting cellular contents
has confirmed that TRAIL-R2 signaling enhances ER stress, leading to apoptosis of MDSCs and a reduction in their immunosuppressive function [116]
