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The oxidative stress in mitochondria could be counteracted by IDH1-driven reductive carboxylation in the cytosol, followed by the transfer of citrate into the mitochondria, which was to be oxidized by IDH2 to generate NADPH [104]

Therefore, these results indicate that the inhibition of NF-B pathway contributes to promote SGNs survived from peroxynitrite injury, and that GPX1 protects SGNs against peroxynitrite-induced damage, at least in part, via blocking the NF-B pathway activation

Tang M, Li Z, Zhang C, Lu X, Tu B, Cao Z, et al

However, the molecular mechanisms of hepatic steatosis occurrence and progression are poorly understood