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We provide compelling evidence showing that impairing the autophagic machinery, results in the accumulation of TG- containing LDs and mitochondria which are unable to efficiently perform FAO and to maintain acetyl-CoA levels, required to support (epigenetic) regulation of PROX1-target genes

Peroxisomes contribute to the acylcarnitine production when the carnitine shuttle is deficient

Treatment area: Face and body

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