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In a mouse model of colorectal cancer, increased arginine levels in the TME promotes tumor development through (i) enhanced angiogenesis due to arginine metabolism and synthesis of the proangiogenic gas nitric oxide (NO) by cells expressing endothelial nitric oxide synthase (eNOS)

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Additionally, while GHK-Cu's mechanisms of action are well-characterized in vitro, more large-scale human trials are needed to confirm its efficacy in diverse populations and conditions

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