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Hence, they act as oxidoreductases that control glutathionylation/deglutathionylation reactions

Using cell lines derived from GBM, sarcoma, and melanoma, we report the unexpected observation that glucose withdrawal induces supraphysiological levels of phosphotyrosine signaling in cells dependent on glucose for viability

Francin-Allami et al

This adduct is formed in at least four times larger quantity than that of 1, N 2 -propano adduct to 2-deoxyguanosine (a DNA adduct) in THP1 monocytes, highlighting its protective action [172]