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Description
Deviations from steady state glutathione redox homeostasis have been discussed as a possible mean to alter the activity of redox-sensitive proteins through switching of critical thiol residues
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Selective molecular alterations in the autophagy pathway in patients with Lewy body disease and in models of -synucleinopathy
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Occluding junction structure-function relationships in a cultured epithelial monolayer
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and progesterone during pregnancy for a successful gestation
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