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The cellular mechanisms by which disruption of astrocytic IGF-1/IGF1R signaling impairs NVC responses likely include dysregulation of metabotropic glutamate receptors, NMDA receptors, and glutamate transporters [14, 17], upregulation of soluble epoxy hydrolase (which eliminates vasodilator EETs produced during astrocyte activation), and altered expression of cytochrome P450 enzymes [14]

Frontiers in Endocrinology , 2020

Tomida M, Mikami I, Takeuchi S, Nishimura H, Akiyama H (2009) Serum levels of nicotinamide N -methyltransferase in patients with lung cancer