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Description
Apoptosis, oxidative and nitrosative stress, mitochondrial malfunction, and uncontrollably high levels of neural inflammation are the results of these alterations
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Results vary based on defect size and individual healing response
10.1016/j.jconrel.2017.03.019 212 ZhangJ.SunY.QuQ.LiB.ZhangL.GuR.et al (2021)
