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Further, it revealed that PI3k inhibitors blocked the enhanced post-synaptic transmission, suggesting this effect might be dependent on PI3k activation [13]

Funding: This work was supported by HHS|NIH|National Heart, Lung, and Blood Institute (NHLBI), R01HL171220, R01HL169203, and R01HL157164, R01HL167846, and R01HL151513
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