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In this example and in type 1 diabetes, it has been discussed whether the normalization of glucose metabolism following rhIGF-I therapy is most importantly associated with insulin-like effects of IGF-I on glucose uptake in muscle or suppression of GH hypersecretion

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In fact, when the Fc portion of daratumumab binds to FcRs on NK cells, cytotoxic proteins (granzymes and perforins) are released, leading to cell death [68]

Distinct from classical GH signaling (experimental): Research suggests minimal stimulation of IGF-1 and limited activation of pathways typically driven by full-length growth hormone