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Thus, while further human trials are essential, it remains to be seen to what extent BPC 157a compound with primarily preclinical support, a wide range of pleiotropic effects, and no observed LD in toxicology studies [3,4,5,6,7,8,9,10,11,12,13,14,16,17,18,19,20,21,22,23,24,27,28,29,30,31,32,34,37,39,40]can truly achieve such outcomes through pharmacological means

Specifically, it decreased the levels of angiogenic factors (CCL20 and GDF15) and immune-suppressive molecules (IL-18BP and TIM-3) while simultaneously boosting the levels of pro-inflammatory cytokines, such as IFN- (17)

Glymphatic Stasis TBI disrupts the polarization of Aquaporin-4 (AQP4) channels, leading to "glymphatic stasis." The brain cannot clear its own waste, leading to the accumulation of toxic proteins (Tau, Amyloid) [7]