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Although the etiopathological mechanisms underlying ADHD are not fully understood, yet the dysregulation in catecholamine neurotransmitters, including dopamine (DA) and norepinephrine (NE) within the prefrontal cortex, hippocampus and striatum, which results in executive dysfunction, had been reported to be tightly linked with the pathogenesis of ADHD (Posner et al
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Immunofluorescence analysis of SLC25A39 in parental and SLC25A39 knockout HeLa cells treated with BSO (1 mM) and erastin (5 uM) for 24 h (bottom)
Interestingly, some of the animal experimental conditions for high-altitude hypobaric hypoxia injury are consistent with the AMS animal model
