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Even intraperitoneal supplementation in peritoneal dialysis fluid has been tested and reported to be associated with improving insulin sensitivity in nondiabetic peritoneal dialysis patients [11]

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administration in a mouse model of lipopolysaccharide-induced AKI proved to mediate against secondary kidney injury by restoring serum levels of urea, creatinine and uric acid, and downregulating the expression of inflammation markers, such as Tnf- and Il-1 [255]

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