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In a model of methotrexate-induced kidney injury, the administration of UMB reduced the expression of inflammation mediators P 38 MAPK and NF-B and the pro-apoptotic molecules BAS and caspase3 while increasing anti-apoptotic levels of the BLC-2 molecule [86]

10.1126/sciadv.abe8290 289 PompeiaS.PanjehS.LouzadaF

Lastly, IN-administered peptides have the potential to directly enter the systemic circulation, avoiding first pass metabolism in the liver, so in certain limited cases peripheral plasma peptide pharmacokinetics have been studied and are well-reviewed here [16,17,22], providing additional insight into the potential for off-target effects [16] and also emphasizing the importance of the local intranasal mechanisms of peptide degradation [23]

Astrocytic glycogen influences axon function and survival during glucose deprivation in central white matter