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10 (17), e018829

Curcumin induced inhibition of AP-1 was a result of its direct interface with AP-1 DNA binding motif ( 3 and NF-B was inhibited by curcumin, leading to the down-regulations of genes involved in apoptosis and cellular proliferation (Mackenzie et al., 2008)

Oxidative stress may contribute to processes involved in cardiovascular disease, including endothelial dysfunction, LDL oxidation, inflammation, mitochondrial injury, and plaque formation
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