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The pathogenesis of MS is attributed to mitochondrial damage and subsequent loss of energy in the CNS of susceptible individuals by oxidative stress, as evidenced by investigations [110,111,112]
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Modulating the LLPS property of intracellular protein condensates will redirect intracellular signals and provide a potential way to regulate cellular physiology
These non-IgE-mediated hypersensitivity reactions are related to complement activation and are commonly referred to as complement activation-related pseudo-allergy (CARPA)
