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Biochem J 1988

The dual roles that are immunostimulatory effect at low concentrations and a suppressive effect at higher concentrations may explain its co-efficacy with IFN- in endometriotic cells 36

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In a mechanistic study with different types of tumour cell lines, the silencing of Grx1 activated the p53 signalling pathway, causing the cell cycle to stop in G1 causing senescence [188], which supported the idea that antioxidant systems, including Grx1, get upregulated in tumour cells as survival mechanism to counteract the high amount of ROS due to the changes in metabolism