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Description
Iron accumulation not only directly damages cardiomyocytes but also exacerbates mitochondrial damage through the promotion of lipid peroxidation (97)
doi: 10.1210/clinem/dgad431 106 KwonTLamsterIBLevinL
In contrast, TIGAR overexpression or treatment with Fer-1, a ferroptosis inhibitor, significantly increases NADPH, GPX4, and GSH levels, reduces GSSG expression, and attenuates neuronal ferroptosis and neurological impairment
FXR, Farnesoid X Receptor
