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This microvascular inflammatory response was also associated with increased vascular permeability to plasma proteins during systemic hypoxia (42)

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In general, our results showed that ethanol exposure may reduce the antioxidant capacity in the hippocampal of WT mice, leading to the occurrence of neuroinflammation, while in APP/PS1 mice, ethanol exposure further worsened this process by exacerbating the pathological progression of AD