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In summary, our findings identify melatonin as a regulator of ILC2 effector function through a glutathione-dependent, receptor-independent pathway
Quantification of fibrosis area was performed using the ImageJ software
Peak mGH levels were significantly higher in ghrelin- and anamorelin-treated animals than in controls, while peak mIGF-1 levels were slightly elevated but not statistically significant
Synergy is inferred from the individual mechanisms, not demonstrated in combination
